50 Best Ocular Hypertension Ad Images in 2020 - BigSpy

Eye Pressure Study
VYZULTA® (latanoprostene bunod ophthalmic solution), 0.024%
In clinical studies, VYZULTA delivered a mean IOP reduction of up to 9.1 mmHg from baseline.[1]* VYZULTA is indicated for the reduction of intraocular pressure (IOP) in patients with open-angle glaucoma or ocular hypertension. IMPORTANT SAFETY INFORMATION • Increased pigmentation of the iris and periorbital tissue (eyelid) can occur. Iris pigmentation is likely to be permanent • Gradual changes to eyelashes, including increased length, increased thickness, and number of eyelashes, may occur. These changes are usually reversible upon treatment discontinuation • Use with caution in patients with a history of intraocular inflammation (iritis/uveitis). VYZULTA should generally not be used in patients with active intraocular inflammation • Macular edema, including cystoid macular edema, has been reported during treatment with prostaglandin analogs. Use with caution in aphakic patients, in pseudophakic patients with a torn posterior lens capsule, or in patients with known risk factors for macular edema • There have been reports of bacterial keratitis associated with the use of multiple-dose containers of topical ophthalmic products that were inadvertently contaminated by patients • Contact lenses should be removed prior to the administration of VYZULTA and may be reinserted 15 minutes after administration • Most common ocular adverse reactions with incidence ≥2% are conjunctival hyperemia (6%), eye irritation (4%), eye pain (3%), and instillation site pain (2%) PLEASE SEE FULL PRESCRIBING INFORMATION AT VYZULTAHCP.COM. *See clinical details at www.vyzulta.com/hcp/clinical-studies. References: 1. Weinreb RN, Scassellati Sforzolini B, Vittitow J, et al. Ophthalmology. 2016;123(5):965-973. 2. VYZULTA Prescribing Information. Bausch & Lomb Incorporated. VYZULTA and the V design are trademarks of Bausch & Lomb Incorporated or its affiliates. ©2020 Bausch & Lomb Incorporated or its affiliates. All rights reserved. VYZ.0106.USA.20
Acurian
High IOP (eye pressure) can damage eyesight over time. Local research studies now enrolling adults with Glaucoma OR Ocular Hypertension. Up to $1000 payment varies by study. Learn more now.
Acurian
Acurian
Acurian
Orbit Academy
Meibomian Gland Dysfunction (MGD) ■■■Risk Factors: Risk factors of MGD include; ■Non ophthalmic: ▪Aging ▪Deficiency of sex hormones notably androgens ▪Other systemic conditions such as rosacea, Sjogren’s syndrome (SS), Stevens-Johnson Syndrome (SJS), psoriasis, atopy, polycystic ovary syndrome (PCOS) and hypertension. ▪Drug-induced; Use of antibiotics, Isotretinoin for Acne, antihistamines, antidepressants, and hormone replacement therapy are found to be associated with MGD. ■Ophthalmic factors: ▪Aniridia ▪Chronic blepharitis ▪Contact lens wear ▪Eyelid tattooing ▪Trachoma ▪Demodex folliculorum infection have been shown to impact Meibomian gland function. ■■■Classification: MGD is classified based on the final consequence of Dry Eye Diseases (DED) into low delivery and high delivery categories based on the secretion status. ■Low delivery status: It is further classified into hyposecretory and obstructive conditions. ▪Hyposecretion is due to decreased Meibomian gland function due to gland atrophy or medications. ▪The obstruction of meibomian glands is the most common cause for hyposecretion which results from the hypertrophy of ductal epithelium and keratinization due to aging, or decreased expression of androgen receptors, or medications. ■Hypersecretory MGD: It results due to excessive secretion of lipids which leads to alterations in tear film. ■■■Diagnosis: The Diagnostic subcommittee at the International Workshop on MGD recommended several diagnostic tests for MGD and proposed two approaches for diagnosing symptomatic MGD-related disease. ■ The diagnostic test sequence for symptomatic MGD-related disease within a General Clinic: ▪Administer symptoms questionnaire ▪Measure blink rate and blink interval ▪Measure lower tear meniscus height ▪Measure tear osmolarity ▪Ocular surface staining: Assess epithelial cell damage. Oxford Grading System, Dry Eye WorkShop (DEWS) grading ▪Break up time *Tear break up time (TBUT): Normal 15-45 seconds *Fluorescein break up time (FBUT):Normal range >10 seconds *Noninvasive break up time (NIBUT): Normal range 40-60 seconds ▪Schirmer test:<5 mm/5 min ▪If MGD (asymptomatic or symptomatic is not diagnosed earlier) *Quantify morphologic lid features: *Expressibility of meibum and its quality *Meibography: Document morphology infra-red or near infra-red video cameras, confocal microscopy, spectral-domain optical coherence tomography (SD-OCT). ■The diagnostic test sequence for symptomatic MGD-related disease within a specialized unit: ▪Symptoms assessment {ocular surface disease index (OSDI) and dry eye questionnaire (DEQ) ▪Measure of osmolarity ▪Tear secretion test ▪Measurement of tear volume ▪Tear evaporation rate (Evaporimetry) ▪Corneal and conjunctival staining ▪Tests to assess ocular inflammation #Orbit_Academy_Oculofacial #Orbit_Academy_Knowledge #Orbit_Academy_Hands_On_Training
Orbit Academy
Meibomian Gland Dysfunction (MGD) ■■■Risk Factors: Risk factors of MGD include; ■Non ophthalmic: ▪Aging ▪Deficiency of sex hormones notably androgens ▪Other systemic conditions such as rosacea, Sjogren’s syndrome (SS), Stevens-Johnson Syndrome (SJS), psoriasis, atopy, polycystic ovary syndrome (PCOS) and hypertension. ▪Drug-induced; Use of antibiotics, Isotretinoin for Acne, antihistamines, antidepressants, and hormone replacement therapy are found to be associated with MGD. ■Ophthalmic factors: ▪Aniridia ▪Chronic blepharitis ▪Contact lens wear ▪Eyelid tattooing ▪Trachoma ▪Demodex folliculorum infection have been shown to impact Meibomian gland function. ■■■Classification: MGD is classified based on the final consequence of Dry Eye Diseases (DED) into low delivery and high delivery categories based on the secretion status. ■Low delivery status: It is further classified into hyposecretory and obstructive conditions. ▪Hyposecretion is due to decreased Meibomian gland function due to gland atrophy or medications. ▪The obstruction of meibomian glands is the most common cause for hyposecretion which results from the hypertrophy of ductal epithelium and keratinization due to aging, or decreased expression of androgen receptors, or medications. ■Hypersecretory MGD: It results due to excessive secretion of lipids which leads to alterations in tear film. ■■■Diagnosis: The Diagnostic subcommittee at the International Workshop on MGD recommended several diagnostic tests for MGD and proposed two approaches for diagnosing symptomatic MGD-related disease. ■ The diagnostic test sequence for symptomatic MGD-related disease within a General Clinic: ▪Administer symptoms questionnaire ▪Measure blink rate and blink interval ▪Measure lower tear meniscus height ▪Measure tear osmolarity ▪Ocular surface staining: Assess epithelial cell damage. Oxford Grading System, Dry Eye WorkShop (DEWS) grading ▪Break up time *Tear break up time (TBUT): Normal 15-45 seconds *Fluorescein break up time (FBUT):Normal range >10 seconds *Noninvasive break up time (NIBUT): Normal range 40-60 seconds ▪Schirmer test:<5 mm/5 min ▪If MGD (asymptomatic or symptomatic is not diagnosed earlier) *Quantify morphologic lid features: *Expressibility of meibum and its quality *Meibography: Document morphology infra-red or near infra-red video cameras, confocal microscopy, spectral-domain optical coherence tomography (SD-OCT). ■The diagnostic test sequence for symptomatic MGD-related disease within a specialized unit: ▪Symptoms assessment {ocular surface disease index (OSDI) and dry eye questionnaire (DEQ) ▪Measure of osmolarity ▪Tear secretion test ▪Measurement of tear volume ▪Tear evaporation rate (Evaporimetry) ▪Corneal and conjunctival staining ▪Tests to assess ocular inflammation #Orbit_Academy_Oculofacial #Orbit_Academy_Knowledge #Orbit_Academy_Hands_On_Training
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